Researchers at National Jewish Health and their colleagues have identified structural and molecular markers in the skin of atopic dermatitis patients that distinguish those who have food allergies from those who do not. The findings strengthen emerging science indicating that the skin plays a major role in food allergies, and suggest that personalized treatments for a subset of atopic dermatitis patients might help prevent food allergies.
The natural history of allergic diseases is referred to as the “atopic march” or the “allergic march.” Allergic diseases can have a big negative impact on a person’s quality of life and can be serious and even life-threatening. So, is there anything we can do to stop the march from progressing?
The atopic march describes how patients who have an allergic disease are more likely to develop another or multiple allergic diseases in their lifetime. This progression commonly starts with eczema in infancy and can develop into food allergy, asthma or hay fever in childhood. Now, new research being presented at the Joint Congress provides evidence that EoE is a late but probable part of this disease progression.
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